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Diabetic retinopathy

A ‘silent disease’ more frightening than the diabetes diagnosis itself

Diabetic retinopathy is a major diabetic complication where high blood sugar damages
retinal vessels and gradually steals vision.

Because early symptoms are rare and hard to notice, if you are diagnosed with diabetes
you need regular eye exams regardless of symptoms.

Types of diabetic retinopathy

Non-proliferative (early-mid)
Non-proliferative

A stage with bleeding, exudates and oedema as retinal microvessels weaken.
Vision loss is mild, but if macular oedema sets in
it can decline sharply,
so regular specialist follow-up and blood-sugar control matter above all.

Proliferative (late)
Proliferative

A high-risk state where poor blood supply
grows abnormal new vessels.
These vessels are fragile and rupture easily, causing vitreous bleed or detachment
that can lead to blindness — fast, precise treatment is required.

Causes of diabetic retinopathy

A major complication in people with diabetes —
the risk rises with blood-sugar control and vascular health.

Diabetic retinopathy causes

Duration of diabetes

The longer you have had diabetes, the higher the rate
(observed in about 60-70%+ of patients with diabetes for 15+ years).

Unstable blood-sugar control

The more unstable your readings, the faster retinal vessel damage progresses.

Other factors

Hypertension, hyperlipidaemia, kidney disease, pregnancy and smoking
affect vascular health and aggravate diabetic retinopathy.

Main diabetic retinopathy symptoms

1

Reduced vision

Vision looks foggy and
blurred as if misted.

2

Floaters

Specks like dust or insects
seem to drift through your view.

3

Metamorphopsia

Objects look distorted
or straight lines bent.

4

Flashes

Flash-like light appears
even with the eyes closed.

5

Field loss

Part of vision is blacked out
or veiled as if by a curtain.

6

Raised eye pressure

May come with eye pain,
headache or nausea.

Diabetic retinopathy treatment

01·04

Medication & monitoring

In early non-proliferative stages we keep blood sugar and pressure under tight control, with regular fundus exams every 3-6 months.

Laser (pan-retinal photocoagulation)

Laser coagulates the peripheral retina, except the photoreceptor-dense centre, to cut oxygen demand and suppress new-vessel growth — lowering bleeding and detachment risk.

Anti-VEGF injections

With macular oedema or when new-vessel suppression is needed, drugs are injected into the eye to reduce swelling and preserve vision (Avastin, Eylea, Lucentis, etc).

Vitrectomy (surgical)

If vitreous bleed does not clear on its own or tractional detachment occurs, surgery directly removes the opaque vitreous and proliferative tissue.